Sandra Bajana
  • Stephenson Cancer Center, University of Oklahoma Health Sciences Center, USA
  • Cancer biology


Ph.D. in Medicine Research, Escuela Superior de Medicina, Mexico, D.F., 2007

Current position

Associate Staff Scientist (Xiao-Hong Sun Lab) at Oklahoma Medical Research Foundation, Foundation, Arthritis and Clinical Immunology Department, Oklahoma City, USA (09/2015-present)


  1. Bajana, S., Turner, S., Paul, J., Ainsua-Enrich, E. and Kovats, S. (2016). IRF4 and IRF8 Act in CD11c+ Cells To Regulate Terminal Differentiation of Lung Tissue Dendritic Cells. J Immunol 196(4): 1666-1677. * Featured in the “In this Issue” section of Feb. 15.
  2. Hu, T., Wang, H., Simmons, A., Bajana, S., Zhao, Y., Kovats, S., Sun, X. H. and Alberola-Ila, J. (2013). Increased level of E protein activity during invariant NKT development promotes differentiation of invariant NKT2 and invariant NKT17 subsets. J Immunol 191(10): 5065-5073.
  3. Bajana, S., Roach, K., Turner, S., Paul, J. and Kovats, S. (2012). IRF4 promotes cutaneous dendritic cell migration to lymph nodes during homeostasis and inflammation. J Immunol 189(7): 3368-3377. *Featured in the “In this Issue” section of the Oct. 1, 201 issue of J. Immunology.
  4. Torres-Aguilar, H., Aguilar-Ruiz, S. R., Gonzalez-Perez, G., Munguia, R., Bajana, S., Meraz-Rios, M. A. and Sanchez-Torres, C. (2010). Tolerogenic dendritic cells generated with different immunosuppressive cytokines induce antigen-specific anergy and regulatory properties in memory CD4+ T cells. J Immunol 184(4): 1765-1775.
  5. Agrawal, H., Jacob, N., Carreras, E., Bajana, S., Putterman, C., Turner, S., Neas, B., Mathian, A., Koss, M. N., Stohl, W., Kovats, S. and Jacob, C. O. (2009). Deficiency of type I IFN receptor in lupus-prone New Zealand mixed 2328 mice decreases dendritic cell numbers and activation and protects from disease. J Immunol 183(9): 6021-6029.
  6. Bajana, S., Herrera-Gonzalez, N., Narvaez, J., Torres-Aguilar, H., Rivas-Carvalho, A., Aguilar, S. R. and Sanchez-Torres, C. (2007). Differential CD4(+) T-cell memory responses induced by two subsets of human monocyte-derived dendritic cells. Immunology 122(3): 381-393.
  7. Figueroa-Vega, N., Galindo-Rodriguez, G., Bajana, S., Portales-Perez, D., Abud-Mendoza, C., Sanchez-Torres, C. and Gonzalez-Amaro, R. (2006). Phenotypic analysis of IL-10-treated, monocyte-derived dendritic cells in patients with systemic lupus erythematosus. Scand J Immunol 64(6): 668-676.
  8. Avila-Moreno, F., Lopez-Gonzalez, J. S., Galindo-Rodriguez, G., Prado-Garcia, H., Bajana, S. and Sanchez-Torres, C. (2006). Lung squamous cell carcinoma and adenocarcinoma cell lines use different mediators to induce comparable phenotypic and functional changes in human monocyte-derived dendritic cells. Cancer Immunol Immunother 55(5): 598-611.
  9. Rivas-Carvalho, A., Meraz-Rios, M. A., Santos-Argumedo, L., Bajana, S., Soldevila, G., Moreno-Garcia, M. E. and Sanchez-Torres, C. (2004). CD16+ human monocyte-derived dendritic cells matured with different and unrelated stimuli promote similar allogeneic Th2 responses: regulation by pro- and anti-inflammatory cytokines. Int Immunol 16(9): 1251-1263.